Methyl jasmonate abrogates rotenone-induced parkinsonian-like symptoms through inhibition of oxidative stress, release of pro-inflammatory cytokines, and down-regulation of immnopositive cells of NF-κB and α-synuclein expressions in mice.

Methyl jasmonate abrogates rotenone-induced parkinsonian-like symptoms through inhibition of oxidative stress, release of pro-inflammatory cytokines, and down-regulation of immnopositive cells of NF-κB and α-synuclein expressions in mice.
Related ArticlesMethyl jasmonate abrogates rotenone-induced parkinsonian-like symptoms through inhibition of oxidative stress, release of pro-inflammatory cytokines, and down-regulation of immnopositive cells of NF-κB and α-synuclein expressions in mice. Neurotoxicology. 2019 Jul 20;: Authors: Alabi AO, Ajayi AM, Ben-Azu B, Bakre AG, Umukoro S Abstract Oxidative stress and neuroinflammation play key roles in the initiation and progression of Parkinson's disease (PD), ... read more
Source: PubMedPublished on 2019-07-24