splitGFP Technology Reveals Dose-Dependent ER-Mitochondria Interface Modulation by α-Synuclein A53T and A30P Mutants.

splitGFP Technology Reveals Dose-Dependent ER-Mitochondria Interface Modulation by α-Synuclein A53T and A30P Mutants.
Related ArticlessplitGFP Technology Reveals Dose-Dependent ER-Mitochondria Interface Modulation by α-Synuclein A53T and A30P Mutants. Cells. 2019 Sep 12;8(9): Authors: Calì T, Ottolini D, Vicario M, Catoni C, Vallese F, Cieri D, Barazzuol L, Brini M Abstract Familial Parkinson's disease (PD) is associated with duplication or mutations of α-synuclein gene, whose product is a presynaptic cytosolic protein also found in mitochondria ... read more
Source: PubMedPublished on 2019-09-25